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In TNBC, enhanced flux through NADPH-generating pathways—such as the pentose phosphate pathway (PPP), malic enzyme 1 (ME1), and cytosolic isocitrate dehydrogenase (IDH1)—has been associated with increased redox buffering capacity and resistance to oxidative stress

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If BPC-157 can promote healing in metabolically compromised tissue, it speaks to the robustness of its mechanisms and suggests broad applicability across patient populations